Interesting idea from 2013/2016, even if only to see how the hypotheses on causes of FM are developing.
This is the original article from 2013 Excessive peptidergic sensory innervation of cutaneous arteriole-venule shunts (AVS) in the palmar glabrous skin of fibromyalgia patients: implications for widespread deep tissue pain and fatigue - PubMed (the link in above thread is dead). Reading it closely shows it is only a small pilot study. In short the relevant 3/4 of the 24 fibro patients had 'more nerves in their hands, which decreases blood flow there (cold temperature) and causes more widespread pain and fatigue in muscles. SNRIs help improve that, that’s why they help some.’ This is FL Rice’s link to the newspaper article quoted in the OP, U of Albany link given by @natria.
His first pubmed-article about this was 2009, but it hasn’t been taken up by anyone for fibro since 2016, a review with PJ Albrecht Fibromyalgia syndrome pathology and environmental influences on afflictions with medically unexplained symptoms - PubMed thinking about if the peripheral nerve alterations discovered have to do with the CSS hypothesis and if it might have to do with estrogen. So maybe his results have invisibly flown into the CSS hypothesis.
Nothing new to date the webpage of his biotech compan Intidyn.
Albrecht has only an article about SFN on pubmed in 2016, both participate in a monkey study for migraine in 2017, and a mouse study for NF1 in 2019.
The defective AV shunts are said in the newspaper version to disturb the transport of nutrition and waste to and from muscles and skin, to disturb temperature regulation and so the nerve fibres, which builds up lactic acid which causes pain & fatigue.
What I identify with in this idea is the Raynaud connection and the fact that I like the others in the thread believe they have a cold body temperature, both of which may not be implied by the research however. What I don’t identify with is the assumption that nerves are hypersensitized, not having hyperalgesia or allodynia for instance. Also my Ache & exhaustibility & needing rest does not feel like the lactic acid from sore muscles, more like flu ache. This doesn’t travel, it’s all over (esp. limbs) at the same time. My single pains, the ones that seemed to move, all had reasons, mainly tendons, which I’ve been able to alleviate bit by bit. Latest one being loins just by putting up with the severe ache from a gym workout. (Still needing help esp. with lower back for sitting & lying down, front for bladder, the rest is stiffness, Ache etc. all over.)
I sometimes wonder whether researchers would be quicker with their ideas if we could describe our pain better (and they would try to listen better).
What it seems tho, is that this idea was not helpful enough to keep interest or that it has been engulfed by the CSS hypothesis anyway, which is still being pursued a lot …